บทคัดย่องานวิจัย

หมวดหมู่ : ฐานข้อมูลงานวิจัย

Understanding the basis of chilling injury in citrus fruit.

Lafuente M.T., Zacarias L., Sala J.M., Sanchez-Ballesta M.T., Lluch, Y., Gosalbes M.J., Granell, A., Marcos J.F., Gonzalez-Candelas L.

5th International Postharvest Symposium . Volume of Abstract . Verona, Italy 6-11 June 2004, p41.

2004

บทคัดย่อ

Understanding the basis of chilling injury in citrus fruit.  Low, non-freezing, temperature is the single most important factor governing maintenance of postharvest quality in stored fruits and vegetables.It may reduce disease, control insect pest, or modify ripening processes but also cause chilling injury (CI).Exposure of chilling-sensitive crops to hardening or nonlethal high temperatures may reduce injury caused by subsequent holding at chilling temperatures.Many varieties of citrus fruit are susceptible to chilling, being ‘ Fortune’ mandarin a good model system to characterise metabolic events underlying CI.

Considerable effort has been directed to understand the physiological and biochemical basis of low temperature tolerance in citrus fruit harvested at different maturity stages, including the role of plant hormones, alteration in lipids, carbohydrate composition, changes in phenylpropanoid metabolism and in oxidative stress-associated processes.However, we still lack solid information o­n the mechanism controlling chilling.The advent of molecular technologies is providing a powerful means for systematically evaluate the expression of large subsets of genes in response to stress cues.Studies of molecular events by which horticultural crops may tolerate chilling are in their early stages but in citrus fruit it has been shown the complexity of molecular mechanism underlying chilling tolerance.

A functional categorisation of genes expressed in heat-conditioned ‘Fortune’ fruit exposed to chilling has revealed that long-term heat-induced chilling tolerance is an active process, which requires new transcription factors, activation of secondary metabolism, and stress-related proteins, while induction of HSPs could be ascribed to the early events of chilling acclimation.Future work is necessary to delineate the precise function of the identified chilling associated responses, but the availability of new molecular strategies will provide an excellent too to continue the study of the mechanism underlying chilling stress.